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MCAT - Biochemistry - Carbohydrate MetabolismLive tournament10 tasks

Gluconeogenesis Tournament

Two onboarding diagrams place gluconeogenesis inside carbohydrate metabolism. Then eight MCAT-DoK rounds: where glucose-6-phosphatase lives, the four bypass enzymes in order, the substrates that actually contribute net carbons, the ATP cost of de novo glucose synthesis, and the Cori-cycle inter-organ loop.

Step 1 of 3 - The bigger pictureGluconeogenesis Tournament

Where the Glucose metabolism (gluconeogenesis branch) fits in Metabolism of carbohydrates

Gluconeogenesis is the reverse of glycolysis through the GLUCOSE METABOLISM hub: liver and kidney cells synthesize free glucose from pyruvate, lactate, alanine, and glycerol during fasting. Click the highlighted Glucose metabolism box to enter the tournament.

G al G6P G1P F ru GOLGI APPARATUS G lycogen synthesis glycogenolysis glycolysis synthesis degradation step-wise degradation gluconeogenesis G lc L ac F6P MONOSACCHARIDES LYSOSOME M an X ul GLYCOGEN XXX/YYY GLYCOGEN METABOLISM XXX/YYY GALACTOSE CATABOLISM XXX/YYY LACTOSE SYNTHESIS PENTOSES NADPH XXX/YYY PENTOSE PHOSPHATE PATHWAY XXX/YYY LYSOSOMAL OLIGOSACCHARIDE CATABOLISM GLUCURONATE XXX/YYY FORMATION OF XYLULOSE-5-PHOSPHATE GA3P DHAP XXX/YYY FRUCTOSE METABOLISM PYRUVATE XXX/YYY GLUCOSE METABOLISM GAGs XXX/YYY GLYCOSAMINOGLYCAN METABOLISM

Click the highlighted Glucose metabolism (gluconeogenesis branch) box to continue.

What this tournament tests

Each task maps to a distinct MCAT cognitive demand. The first two orient you in the broader topology; the next eight test the high-yield mechanism, regulation, sequence and quantitative reasoning that consistently appear on test day.

1

The Bigger Picture

Locate gluconeogenesis inside Reactome's Metabolism of Carbohydrates overview.

2

Whole-Pathway Overview

Pan and zoom the WikiPathways gluconeogenesis figure before you play.

3

Fill in the Blank

Locate glucose-6-phosphatase in the ER lumen of liver / kidney / intestine.

4

Disruptor

Walk the Von Gierke quartet: hypoglycemia, hepatomegaly, lactic acidosis, hyperuricemia.

5

Sequence Ordering

Walk the four bypass enzymes from pyruvate to free glucose in order.

6

Match the Pairs

Pair each gluconeogenic substrate with the metabolic context that delivers it.

7

Numeric Input

Compute the 6 ATP equivalents per glucose synthesized de novo from 2 pyruvate.

8

Select All That Apply

Identify which substrates can serve as a NET source of glucose carbons.

9

Odd One Out

Distinguish the gluconeogenic bypass enzyme from the irreversible glycolytic ones.

10

Cori Cycle

Recognize the muscle-liver lactate-glucose loop and its alanine-shuttle cousin.

Public leaderboard

Your score posts to a global, persistent leaderboard scored by points first, time as tiebreaker.

Gluconeogenesis in 60 seconds

Gluconeogenesis is the liver and kidney's synthesis of free glucose from non-carbohydrate precursors during fasting. It uses most of the glycolytic enzymes in reverse, plus four irreversible bypass enzymes: pyruvate carboxylase (mitochondrial, biotin-dependent, requires acetyl-CoA), PEPCK (cytosolic, GTP), fructose-1,6-bisphosphatase (cytosolic), and glucose-6-phosphatase (ER lumen of liver / kidney / intestine only).

Skeletal muscle has no glucose-6-phosphatase, which is why it cannot release free glucose to blood. Glucagon drives gluconeogenesis (via cAMP -> PKA -> ACC + glycogen-synthase phosphorylation, and via low F2,6BP); insulin opposes it.

Substrates: lactate (Cori cycle from anaerobic muscle / RBCs), alanine (alanine cycle - amino group carrier from peripheral protein breakdown), glycerol (from adipose lipolysis, enters at DHAP), and propionyl-CoA (from odd-chain FA / branched-chain AAs - the only gluconeogenic fatty-acid contribution).

The big trap: acetyl-CoA cannot become net glucose in mammals. Each turn of the TCA loses 2 C as CO2 before any new OAA is made. So even-chain fatty acids and ketogenic amino acids cannot be net glucose precursors.

FAQ

Why does Von Gierke disease cause hyperuricemia?

Glucose-6-phosphatase deficiency lets G6P accumulate. Excess G6P shunts into the PPP -> ribose-5-P -> nucleotides -> purine catabolism -> uric acid. So Von Gierke is the textbook 'metabolic gout' picture in pediatrics.

Can I make glucose from fat?

Almost never in mammals. Even-chain fatty acids -> all carbons end up as CO2 in the TCA before glucose can be made. Only the propionyl-CoA fragment from ODD-chain fatty acids (or branched-chain AAs) escapes the TCA carbon-loss trap and contributes net carbons via succinyl-CoA.

Do I need an account to play?

No. The tournament is fully public. You get a randomized handle and your score posts to the public leaderboard at the bottom of this page.